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For example, accessing the descriptor data of each subclass is performed with the non-virtual Ptr() method defined in the TDesC base class (In fact, there are no virtual methods within the descriptor hierarchy) This method uses the type data stored in each descriptor instance as the control variable for a switch statement that then returns the proper address referring to the beginning of the data By preserving the length (and maximum length) of a descriptor, its method calls are able to perform appropriate validation and prevent the unintentional access to out-of-bounds memory When a method call would read or write beyond the data boundaries, the process will panic and immediately terminate This means that with traditional descriptors, the burden of correct memory management falls solely on the shoulders of the developer They must verify that the descriptor can accommodate the amount of incoming data before calling a method that would modify the contents of the descriptor Recognizing these difficulties, Symbian has developed and released a new library The EUserHL Core Idioms Library provides a pair of descriptors (LString for Unicode-aware text and LData for simple byte buffers) that manage their own memory These descriptors automatically reallocate themselves to increase their capacity and then release the resources when they go out of scope, similar to the behavior of standard C++ strings Because these two classes have been derived from their respective TDesC class (TDesC8 and TDesC16), they can be used as parameters

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Type the following sentence and save the le in the My Documents folder as Command Line Testtxt (see Figure 13-6): This is a small sentence of text created in Notepad that I will visit again in the Lab Exercises in the next chapter, Working with the Command-Line Interface

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Disorders of neuromuscular transmission (DNMT), particularly myasthenia gravis (MG), represent the prize pupils of neuromuscular clinicians (Table 23 1) Most of these disorders have either an autoimmune or a toxic etiology, the latter resulting from either infectious or other biological sources MG is the prototypic autoimmune disease Its study has provided insights into its biology as well as of other disorders of autoimmunity The basic science underlying the pathology and pathophysiology of MG is conceptualized better than virtually any other neuromuscular disorder As MG is an eminently manageable disorder in the majority of cases, both patient and physician are grati ed by its identi cation and treatment In many cases, treatment of MG and other DNMT represent the effective translation of basic science to the bedside The historical highlights of DNMT can be summarized as follows The initial insights were physiological The concept that neuromuscular function is mediated electrically was initially promoted in the late eighteenth century and championed by Galvania and Matteucci That transduction of an electrical to a chemical signal served as a mediator of neuromuscular transmission (NMT) was most notably promoted by Bernard, Vulpian, and Langley through the eighteenth and early nineteenth century Their deductions arose from observations made by the actions of the neuromuscular blocking agent curare and the stimulant nicotine Descriptions of the anatomy of the neuromuscular junction (NMJ) began with the advent of light microscopy in the mid-nineteenth century and have been further expanded by electron and confocal laser scanning microscopy Doyere, Ranvier, Tello, Cajal, and Couteaux are notable contributors to our knowledge, the latter credited with insights into junctional architecture1 Thomas Willis, a seventeenth-century physiologist, is frequently credited for initially describing the clinical syndrome of MG Erb, Oppenheim, and Gold am are commonly credited with putting it on the map during the last two decades of the nineteenth century The relevance of junctional anatomy to MG came into focus in the 1930s, when Mary Walker discovered and described the bene ts of cholinesterase inhibitors in these patients In 1960, Simpson rst proposed that MG was an autoimmune disease resulting from an antibody-mediated blockade of NMT Daniel Drachman and colleagues took advantage of radiolabeled -bungarotoxin in the early 1970s to demonstrate that antiacetylcholine receptor (AChR) numbers were reduced in patients with MG Within a few years, Lindstrom and colleagues were instrumental in identifying the AChR antibodies proposed by Simpson This chapter will review the clinical, pathophysiological, and pathological characteristics of, as well as the diagnostic and management issues pertaining to, the more common or notable pre- and postsynaptic DNMT.

directly when accessing the preexisting platform API LString and LData should be used instead of any of the other descriptor classes

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